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PRODID:-//Memento EPFL//
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SUMMARY:Humoral control of mitochondrial function and longevity
DTSTART:20111209T160000
DTSTAMP:20260511T062159Z
UID:6bc5c55ef2206c9ae72055d8082416725054d52fce60db4cd70f3c6f
CATEGORIES:Conferences - Seminars
DESCRIPTION:Dr. Andrew Dillin\n"Humoral control of mitochondrial function 
 and longevity"\nThe endosymbiotically-derived relationship between the mit
 ochondria and cell initially provided the cell with the energy necessary t
 o allow for differentiation and to eventually enter into a state of comple
 xity in which specialized organs and tissues could evolve. Two billion yea
 rs later\, whole organisms composed of complex networks of organs\, tissue
 s\, and cells\, are utterly dependent upon mitochondria for their energeti
 c functions. In the face of an increasingly-complex environment\, the euka
 ryotic cell now spends a considerable amount of mitochondrial-derived ener
 gy in an attempt to coordinate homeostasis and to minimize the potential f
 or stochastic events to disrupt whole organism function. In consideration 
 of this context\, it seems logical that changes to the health of the mitoc
 hondria might initiate a signal that would need to be communicated to the 
 whole organism. Certainly in mitotic tissues\, mitochondria are essential 
 for the initiation of the apoptotic pathways\, and mitochondria are known 
 to help signal metabolic states between tissues as well. But can mitochond
 ria in one cell communicate with and affect mitochondrial function in dist
 al cells? If so\, how do the distal mitochondria hear and respond to such 
 a signal?
LOCATION:SV 1717a
STATUS:CONFIRMED
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