retrieve:
Return the details about the given Event id.

list:
List all Event objects.

GET /api/v1/events/?format=api&offset=190&ordering=event__url_online_room
HTTP 200 OK
Allow: GET, HEAD, OPTIONS
Content-Type: application/json
Vary: Accept

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            "description": "<strong>A Lola and John Grace Distinguished Lecture in Cancer Research</strong>\r\n<div class=\"elementor-element elementor-element-98cefd8 elementor-widget elementor-widget-heading\">\r\n<div class=\"elementor-widget-container\">\r\n<div class=\"is-active tabs-panel\">\r\n<div class=\"bundle--tabs paragraph paragraph--type--tabs paragraph--view-mode--default\">\r\n<div class=\"row\">\r\n<div class=\"columns small-12\">\r\n<div class=\"body field__item\">David Pellman, M.D. is the Margaret M. Dyson Professor of Pediatric Oncology at the Dana-Farber Cancer Institute, a Professor of Cell Biology at Harvard Medical School, an Investigator of the Howard Hughes Medical Institute, and the Associate Director for Basic Science at the Dana-Farber/Harvard Cancer Center.  He received his undergraduate and medical degrees from the University of Chicago.  During medical school, he did research at the Rockefeller University.  His postdoctoral fellowship was at the Whitehead Institute/Massachusetts Institute of Technology.<br>\r\n<br>\r\nThe Pellman Lab works on the mechanism of cell division and how certain cell division errors drive rapid genome evolution.  His research has contributed to the understanding of cell division, the origin of cell division errors in cancer, how cell division errors drive genetic instability and the mechanisms driving rapid evolution of cancer genomes. Our contributions include (1) the co-discovery of formin-dependent actin nucleation and a mechanism for spindle positioning during asymmetric cell division, (2) discoveries establishing that whole genome duplication can drive tumorigenesis, alter cell physiology, and accelerate evolutionary adaptation, and (3) the discovery of a mechanism explaining chromothripsis, a mutational process that generates rapid karyotype evolution in cancer and congenital disease.</div>\r\n</div>\r\n</div>\r\n</div>\r\n</div>\r\n</div>\r\n</div>\r\n\r\n<div class=\"elementor-element elementor-element-e552a16 elementor-widget elementor-widget-text-editor\">\r\n<div class=\"elementor-widget-container\"> </div>\r\n</div>",
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            "organizer": "Prof. Pierre Gönczy",
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            "id": 71840,
            "title": "Precision Oncology Beyond Genomics: Making the Case in Pediatric Leukemia",
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            "description": "<strong>A Lola and John Grace Distinguished Lecture in Cancer Research</strong>\r\n<div class=\"elementor-element elementor-element-98cefd8 elementor-widget elementor-widget-heading\">\r\n<div class=\"elementor-widget-container\"><br>\r\nDr. Jun J. Yang has a long-standing interest in human genetics, dating back to his early education in China where he received his undergrad and master’s degrees. He completed his PhD training at Purdue University and moved to St. Jude where he transitioned from postdoc training to faculty in 2010. Dr. Yang currently serves as Vice Chair for the Department of Pharmacy and Pharmaceutical Sciences and is the Associate Director of the Hematological Malignancies T32 Training Program at the St. Jude Comprehensive Cancer Center. He was elected President of the Pharmacogenomics Research Network in 2021. Dr. Yang has been continuously funded by the NIH since 2011 and receives funding from prestigious nonprofit organizations including the V Foundation. Dr. Yang is committed to genomic and pharmacogenomic research of pediatric leukemia and leverages multi-disciplinary collaborations to ask scientific questions on a global scale.</div>\r\n</div>\r\n\r\n<div class=\"elementor-element elementor-element-e552a16 elementor-widget elementor-widget-text-editor\">\r\n<div class=\"elementor-widget-container\"> </div>\r\n</div>",
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